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T +27 (0)21 900 6277
E reception@haemalife.co.za

A 4G Riverside View Chambers
Netcare Kuils River Hospital
33 Van Riebeeck Road
Kuils River
7580


Get In Touch

Come and visit us or simply send us an
email anytime you want.

CONTACT INFO

Reception:
reception@haemalife.co.za

Bookings:
bookings@haemalife.co.za

Accounts:
accounts@haemalife.co.za

Medical Aid authorization:
casemanagement@haemalife.co.za

Transplant information:
maryna.delange@haemalife.co.za

Chemotherapy information:
ina@haemalife.co.za

Pharmacy information:
pharmacy@haemalife.co.za

Social Worker:
socialwork@haemalife.co.za

Practice Manager:
carl.toua@haemalife.co.za

Office Hours

Monday – Thursday

7:30am – 4pm

Friday

7:30am – 12pm

Saturday – Sunday

Closed

Public Holidays

Closed

Contact Us

T
+27 (0)21 900 6277
E
reception@haemalife.co.za

A

4G Riverside View Chambers
Netcare Kuils River Hospital
33 Van Riebeeck Road
Kuils River
7580

CONTACT INFO

Reception:

reception@haemalife.co.za

 

Bookings:
bookings@haemalife.co.za

 

Accounts:
accounts@haemalife.co.za

 

Authorization:
casemanagement@haeamalife.co.za

 

Social Work:
socialwork@haemalife.co.za

 

Transplant information:
maryna.delange@haemalife.co.za

 

Chemotherapy information:
ina@haemalife.co.za

 

Pharmacy information:
pharmacy@haemalife.co.za

 

Practice Manager:
carl.toua@haemalife.co.za

Practising Hours

Mon – Thu       8 am – 7pm
Fri             8 am – 7pm

Sat – Sun          Closed

Contact Us

T    +27 (0)21 518 0717
F    +27 (0)21 900 6323
E    reception@haemalife.co.za

A

4G Riverside View Chambers Netcare Kuils River Hospital 33 Van Riebeeck Road
Kuils River
7580

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CLL

Chronic Lymphocytic Leukemia (CLL) is a slow-growing cancer of B-lymphocytes, a type of white blood cell. In CLL, abnormal B-cells accumulate in the blood, bone marrow, and lymphoid tissues, often crowding out healthy cells and weakening the immune system.

Diagnosis is made through blood tests showing a high number of mature-looking lymphocytes, flow cytometry to confirm their identity, and sometimes bone marrow biopsy. Genetic testing helps predict disease course and guide treatment.

Types of CLL are classified mainly by genetic features (e.g., deletions of 17p, 13q, or mutations like TP53) and immunoglobulin heavy chain (IGHV) mutation status, which influence prognosis and therapy choice.

Treatment options depend on symptoms, disease progression, and risk factors. Many patients are initially monitored without treatment (“watch and wait”). When therapy is needed, options include targeted therapies (BTK inhibitors, BCL2 inhibitors), chemotherapy, monoclonal antibodies, and in select cases, stem cell transplantation. Supportive care focuses on managing infections and other complications.

MYELOMA

Multiple Myeloma (MM) is a cancer of plasma cells, a type of white blood cell that produces antibodies. In MM, abnormal plasma cells accumulate in the bone marrow, crowding out healthy cells and producing defective antibodies, which can cause organ damage.

Diagnosis involves blood tests (showing abnormal proteins or anemia), urine tests (detecting excess light chains), and a bone marrow biopsy. Imaging like X-rays, MRI, or PET/CT scans can detect bone lesions. Additional tests identify genetic changes that affect prognosis.

Types of Myeloma are often classified based on the type of abnormal protein produced (IgG, IgA, light-chain, etc.) and genetic features of the plasma cells. Smoldering (asymptomatic) myeloma is an early, slow-growing form.

Treatment options depend on age, health, and disease stage. Common approaches include chemotherapy, targeted therapies (like proteasome inhibitors or immunomodulators), immunotherapy (monoclonal antibodies), stem cell transplantation, and supportive care to manage bone disease, anemia, or kidney problems.

ALL

Acute Lymphoblastic Leukemia (ALL) is a fast-growing cancer of the blood and bone marrow in which immature lymphoid cells (B or T lymphocytes) multiply uncontrollably, crowding out normal blood cells and impairing immune function.

Diagnosis involves blood tests showing abnormal lymphoblasts, followed by a bone marrow biopsy. Immunophenotyping, cytogenetic, and molecular testing are used to confirm the diagnosis, classify the subtype, and assess prognosis.

Types of ALL are classified based on the lymphocyte involved (B-cell or T-cell ALL) and specific genetic abnormalities. Certain subtypes, such as Philadelphia chromosome–positive ALL, have important treatment implications.

Treatment options depend on age, risk factors, and genetic findings. Therapy typically includes multi-phase chemotherapy, targeted therapies (such as tyrosine kinase inhibitors), immunotherapy (including monoclonal antibodies or CAR T-cell therapy), and in some cases stem cell transplantation. Supportive care is essential throughout treatment.

AML

Acute Myeloid Leukemia (AML) is a fast-growing cancer of the blood and bone marrow in which abnormal myeloid cells multiply and crowd out normal blood cells, leading to anemia, infections, and bleeding.

Diagnosis is made through blood tests showing abnormal cells, followed by a bone marrow biopsy. Additional tests such as cytogenetic, molecular, and immunophenotyping studies help confirm AML and guide treatment.

Types of AML are classified based on cell appearance and genetic changes. Major systems include the WHO and FAB classifications. Subtypes are defined by specific chromosome or gene abnormalities, such as acute promyelocytic leukemia (APL), which requires specialized therapy.

Treatment options depend on patient age, health, and AML subtype. They may include intensive chemotherapy, targeted therapies (such as FLT3 or IDH inhibitors), immunotherapy, and stem cell (bone marrow) transplantation. Supportive care, including antibiotics and blood transfusions, is also essential.